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Gene expression profile analyses of cortical dysplasia by cDNA arrays

• Seung Ki Kim, K C Wang, +8 authors B. -K. Cho
• Epilepsy Research
• 2003

Cortical dysplasia (CD) is a well-recognized cause of intractable epilepsy, especially in children and is characterized histologically by derangements in cortical development and organization. The… (More)

Radiation-induced cerebellar glioblastoma at the site of a treated medulloblastoma: case report.

• Seung-Yeob Yang, K C Wang, +5 authors S. -K. Kim
• Journal of neurosurgery
• 2005

Radiation-induced glioblastoma multiforme (GBM) is a rare complication of radiotherapy. The authors report such a case occurring 10 years after treatment of cerebellar medulloblastoma. The patient… (More)

Plasma matrix metalloproteinases, cytokines and angiogenic factors in moyamoya disease.

• H S Kang, Jin Hyun Kim, +5 authors S. -K. Kim
• Journal of neurology, neurosurgery, and…
• 2010

OBJECTIVE To document the expression patterns of various matrixins, cytokines and angiogenic factors in plasma to assess their involvement in the pathogenesis of moyamoya disease (MMD). METHODS… (More)

Smooth-muscle progenitor cells isolated from patients with moyamoya disease: novel experimental cell model.

• H S Kang, Youn-Joo Moon, +7 authors S. -K. Kim
• Journal of neurosurgery
• 2014

OBJECT Moyamoya disease (MMD) is a cerebrovascular occlusive disease affecting bilateral internal carotid termini. Smooth-muscle cells are one of the major cell types involved in this disease… (More)

Single nucleotide polymorphisms of tissue inhibitor of metalloproteinase genes in familial moyamoya disease.

• H S Kang, S. -K. Kim, B. -K. Cho, Yong-in Kim, Yong-Seung Hwang, K C Wang
• Neurosurgery
• 2006

OBJECTIVE The genes encoding tissue inhibitor of metalloproteinase (TIMP) 4 and TIMP2 span chromosomes 3p24.2-p26 and 17q25, respectively, which are the locations of familial moyamoya disease (FMMD)… (More)

Overexpression of cyclooxygenase-2 in childhood ependymomas: role of COX-2 inhibitor in growth and multi-drug resistance in vitro.

• Seung Ki Kim, Su-Young Lim, +5 authors B. -K. Cho
• Oncology reports
• 2004

The management of ependymomas remains one of the most frustrating issues in pediatric neuro-oncology. Gross total resection is not always possible, and intensive chemotherapy and craniospinal… (More)

Tissue expression of manganese superoxide dismutase is a candidate prognostic marker for glioblastoma.

• Chul-Kee Park, Ji Hye Jung, +8 authors B. -K. Cho
• Oncology
• 2009

BACKGROUND Characterization of a rare subgroup of glioblastoma patients who survive for more than 3 years (long-term survival glioblastoma, LTSGBL, patients) may be helpful to identify prognostic… (More)

Interaction between p53 and p16 expressed by adenoviral vectors in human malignant glioma cell lines.

• Seung Ki Kim, K C Wang, +5 authors H J Kim
• Journal of neurosurgery
• 2002

OBJECT Multiple gene replacements have been examined as a potential treatment modality for malignant gliomas. Nevertheless, no reports are available that detail the synergy, additivity, or antagonism… (More)

CKD-602, a camptothecin derivative, inhibits proliferation and induces apoptosis in glioma cell lines.

• Yong-in Kim, Chul-Kee Park, +5 authors B. -K. Cho
• Oncology reports
• 2009

CKD-602 7-[2-(N-isopropylamino)ethyl]-(20S)-camptothecin, belotecan) is a synthetic water-soluble camptothecin derivative and topoisomerase inhibitor that has been shown to have clinical anticancer… (More)

Adenoviral p16/CDKN2 gene transfer to malignant glioma: role of p16 in growth, invasion, and senescence.

• Seung Ki Kim, K C Wang, +7 authors H J Kim
• Oncology reports
• 2003

In gliomas, a high frequency of homozygous p16 gene deletions have been demonstrated, which are believed to be linked with malignant progression. The aim of this study was to assess the role of p16… (More)

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