Xingshui Cao

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Mitochondrial dysfunction has been implicated in the pathogenesis of Alzheimer’s disease (AD). However, it is obscure how amyloid-beta (Aβ) can impair mitochondria in the early stage of AD pathology. Using PrP-hAPP/hPS1 double-transgenic AD mouse model, we find that abnormal mitochondrial morphology and damaged mitochondrial structure in hippocampal neurons(More)
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