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Tumor necrosis factor-alpha contributes to below-level neuropathic pain after spinal cord injury.
OBJECTIVE Our objective was to elucidate the mechanisms responsible for below-level pain after partial spinal cord injury (SCI). METHODS We used lateral hemisection to model central neuropathicExpand
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HSV-mediated transfer of interleukin-10 reduces inflammatory pain through modulation of membrane tumor necrosis factor α in spinal cord microglia
To dissect the molecular basis of the neuroimmune response associated with the genesis of inflammatory (nociceptive) pain, we constructed a herpes simplex virus-based gene transfer vector to expressExpand
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Release of GABA from sensory neurons transduced with a GAD67-expressing vector occurs by non-vesicular mechanisms
We have demonstrated that dorsal root ganglion neurons transduced with a recombinant replication-defective herpes simplex virus vector coding for glutamic acid decarboxylase (QHGAD67) release GABA toExpand
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