Weisong Zhou

Kasia Goleniewska4
R Stokes Peebles3
4Kasia Goleniewska
3R Stokes Peebles
3Shinji Toki
2R. Stokes Peebles
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In this paper, input-to-state stability problems for a class of recurrent neural networks model with multiple time-varying delays are concerned with. By utilizing the Lyapunov–Krasovskii functional method and linear matrix inequalities techniques, some sufficient conditions ensuring the exponential input-to-state stability of delayed network systems are(More)
Respiratory syncytial virus (RSV) is associated with wheezing and childhood asthma. We previously reported that RSV infection prolongs methacholine-induced airway hyperresponsiveness in ovalbumin (OVA)-sensitized mice. In addition, allergically sensitized RSV-infected (OVA/RSV) mice had more abundant airway epithelial mucus production compared with OVA mice(More)
  • Rinat Zaynagetdinov, Georgios T. Stathopoulos, Taylor P. Sherrill, Dong-Sheng Cheng, Allyson G. McLoed, Jamie A. Ausborn +8 others
  • 2012
The mechanisms by which chronic inflammatory lung diseases, particularly chronic obstructive pulmonary disease, confer enhanced risk for lung cancer are not well-defined. To investigate whether nuclear factor (NF)-κB, a key mediator of immune and inflammatory responses, provides an interface between persistent lung inflammation and carcinogenesis, we(More)
  • Weisong Zhou, Dustin R. Dowell, Matthew M. Huckabee, Dawn C. Newcomb, Madison G. Boswell, Kasia Goleniewska +10 others
  • 2012
BACKGROUND Prostaglandin I(2) (PGI(2)), a lipid mediator currently used in treatment of human disease, is a critical regulator of adaptive immune responses. Although PGI(2) signaling suppressed Th1 and Th2 immune responses, the role of PGI(2) in Th17 differentiation is not known. METHODOLOGY/PRINCIPAL FINDINGS In mouse CD4(+)CD62L(+) naïve T cell culture,(More)
  • Shinji Toki, Kasia Goleniewska, Sara Reiss, Weisong Zhou, Dawn C Newcomb, Melissa H Bloodworth +5 others
  • 2016
BACKGROUND Group 2 innate lymphoid cells (ILC2) are an important source of the type 2 cytokines interleukin (IL)-5 and IL-13 that are critical to the allergic airway phenotype. Previous studies reported that histone deacetylase (HDAC) inhibition by trichostatin A (TSA) downregulated adaptive allergic immune responses; however, the effect of HDAC inhibition(More)
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