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Acetylcysteine for prevention of acute deterioration of renal function following elective coronary angiography and intervention: a randomized controlled trial.
TLDR
Acetylcysteine protects patients with moderate chronic renal insufficiency from contrast-induced deterioration in renal function after coronary angiographic procedures, with minimal adverse effects and at a low cost. Expand
Effects of hyperglycemia and hyperinsulinemia on satiety in humans.
TLDR
The study suggests that in humans hyperglycemia induces satiety, and this effect seems not to be mediated by insulin, since HI had no effect on appetite. Expand
Effect of insulin on basal and cholecystokinin-stimulated gallbladder motility in humans.
TLDR
In healthy subjects acute hyperglycemia and euglycemic hyperinsulinemia reduce basal duodenal bilirubin output and inhibit gallbladder emptying stimulated by low dose cholecystokinin, and results suggest that insulin is involved in the inhibitory effect of hyper glycemia on basal and choleCystokerin-stimulated gallbladders motility. Expand
Effect of hyperglycaemia on gallbladder motility in Type 1 (insulin-dependent) diabetes mellitus
TLDR
It is concluded that during euglycaemia the gallbladder contraction in response to cholecystokinin in Type 1 diabetic patients is not significantly different from control subjects. Expand
Effect of hyperglycemia on gastric acid secretion and gastrin release induced by intravenous amino acids.
TLDR
It is concluded that IVAAs stimulate gastric acid secretion, and gastrin and PP release; acute hyperglycemia reduces IVAA-stimulated acid output, and gastroenterological hormone secretion; and circulating nutrients influence gastric Acid and gastrointestinal hormone secretion. Expand
Hyperglycemia modulates gallbladder motility and small intestinal transit time in man
TLDR
It is concluded that low doses of intraduodenal emulsified fat result in significant gallbladder contraction and CCK secretion, and acute hyperglycemia inhibits basal and stimulated plasma PP secretion, suggesting impaired vagal-cholinergic tone during hyper glycemia. Expand
Effect of bombesin on esophageal motility in humans.
TLDR
The effect of bombesin on esophageal motility is not dependent on vagal cholinergic mechanisms and is not mediated by the action of gastrointestinal hormones released byBombesin. Expand
The role of cholecystokinin and the cholinergic system in intravenous amino acid-induced gallbladder emptying.
TLDR
In healthy volunteers intravenous infusion of high doses of amino acids results in a significant gallbladder contraction, which is inhibited by CCK-A receptor blockade and by atropine. Expand
Effect of acute hyperglycemia on esophageal motility and lower esophageal sphincter pressure in humans.
TLDR
It is concluded that blood glucose levels affect esophageal motility, acute hyperglycemia reduces LESP and impairs esophages motility under both basal and edrophonium-stimulated conditions, and hyperglyCEmia reduces plasma PP levels, suggesting impaired vagal-cholinergic activity during hyperglycermia. Expand
Hyperglycemia reduces gastric secretory and plasma pancreatic polypeptide responses to modified sham feeding in humans.
TLDR
It is indicated that (1) serum glucose affects cephalic-stimulated gastric acid secretion, and (2) PP secretion after MSF is significantly reduced during hyperglycemia suggesting impaired vagal-cholinergic activity during hyper glycemia. Expand
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