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We propose the theory that prolonged cerebral vasospasm involves three phases: (1) the initial muscular contraction of the arterial wall; (2) a secondary injury to the artery that consists of endothelial desquamation with adherence of platelets to te denuded internal elastic lamina and mural thrombus formation; and (3) the repair process, which is the(More)
The records of 104 patients treated prophylactically with heparin during gradual carotid ligation after rupture of an intracranial aneurysm, 11 patients, who received heparin after the development of an ischemic neurological deficit during clamp closure, and 46 patients who did not receive heparin during clamp closure were reviewed. The incidence of(More)
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