V. Russell

Publications164
h-index38
Citations6,584
Highly Influential Citations339
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A dynamic developmental theory of attention-deficit/hyperactivity disorder (ADHD) predominantly hyperactive/impulsive and combined subtypes.
TLDR
The dynamic developmental behavioral theory describes how individual predispositions interact with these conditions to produce behavioral, emotional, and cognitive effects that can turn into relatively stable behavioral patterns.
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Rodent Models of Attention-Deficit/Hyperactivity Disorder
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Animal models of attention-deficit hyperactivity disorder
TLDR
Animal models of ADHD suggest that the dopaminergic system is functionally impaired, and evidence obtained from animal models suggests that psychostimulants may not be acting on the dopamine transporter to produce the expected increase in extracellular dopamine concentration in ADHD.
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The Conscious Perception of the Sensation of Fatigue
TLDR
It is suggested that the sensation of fatigue is the conscious awareness of changes in subconscious homeostatic control systems, and is derived from a temporal difference between subconscious representations of these homeostatics in neural networks that are induced by changes in the level of activity.
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Neurobiology of animal models of attention-deficit hyperactivity disorder
  • V. Russell
  • Biology, Psychology
    Journal of Neuroscience Methods
  • 15 April 2007
View on Elsevier
Hypodopaminergic and hypernoradrenergic activity in prefrontal cortex slices of an animal model for attention-deficit hyperactivity disorder — the spontaneously hypertensive rat
  • V. Russell
  • Biology, Psychology
    Behavioural Brain Research
  • 10 March 2002
View on Elsevier
The spontaneously hypertensive rat model of ADHD – The importance of selecting the appropriate reference strain
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Differences between electrically-, ritalin- and d-amphetamine-stimulated release of [3H]dopamine from brain slices suggest impaired vesicular storage of dopamine in an animal model of…
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Response variability in Attention-Deficit/Hyperactivity Disorder: a neuronal and glial energetics hypothesis
TLDR
The hypothesis that in ADHD, astrocyte function is insufficient, particularly in terms of its formation and supply of lactate is proposed, extending existing theories of ADHD by proposing a physiological basis for specific aspects of the ADHD phenotype.
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Origins of altered reinforcement effects in ADHD
TLDR
Theoretical and experimental analyses of these moderating factors will help to determine just how reinforcement processes are altered in ADHD, and can only help to improve treatment strategies for ADHD.
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