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[Endotoxin and the polymorphonuclear leukocyte system].
Due to its cell pool autoturnover, gram-negative intestinal microflora releasing the biologically active agent lipopolysaccharide may cause portal and systemic endotoxemias. Lipopolysaccharide mayExpand
[Interaction between the polymorphonuclear leukocyte system and bacterial agents].
TLDR
Peculiarities of interaction between the polynuclear leucocyte (PNL) system and bacterial agents are considered within the framework of four typical clinico-biological situations, characterized by a purulent inflammatory-infectious process. Expand
Electron-microscopic autoradiography of RNA synthesis in the injured myocardium
TLDR
A burn of the wall of the left ventricle was produced in newborn rats and the density of distribution of silver grains above the nucleus and cytoplasm of the cardiomyocytes was lower in the experimental animals than in the controls. Expand
[CLearance of Pseudomonas aeruginosa from the lungs in experimental burn trauma].
TLDR
Reduced general antibacterial resistance as a result of burn facilitates rapid penetration of P. pyocyanea into the lymph nodes, blood, kidneys, liver, spleen where it is found 6 hours after infection. Expand
[Acute respiratory distress syndrome in endotoxic shock].
TLDR
Large amounts of lipopolysaccharides released in blood after death of Gram-negative bacteria are responsible for endotoxinemia and endotoxic shock and their transfer by polymorphonuclear leukocytes is one of the mechanisms. Expand
[Teseus phenomenon: a new symptom].
Morphological proof of a new phenomenon is presented: moderately activated phagocyte (alveolar macrophage, neutrophil leucocyte) remains connected with a site of its movement by a thin thread. TheExpand
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