Tsuyoshi Toyokawa

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To delineate the in vivo cardiac functions requiring normal delta protein kinase C (PKC) activity, we pursued loss-of-function through transgenic expression of a deltaPKC-specific translocation inhibitor protein fragment, deltaV1, in mouse hearts. Initial results using the mouse alpha-myosin heavy chain (alphaMHC) promoter resulted in a lethal heart failure(More)
  • GERALD W. DORN, ULRIKE DELLING, EVANGELIA KRANIAS, BRUCE J. ARONOW, TSUYOSHI TOYOKAWA, AMY CANNING +8 others
  • 2000
24 times a year (twice monthly) by the American Physiological Society, 9650 Rockville Pike, Bethesda MD systems with techniques linking genes and pathways to physiology, from prokaryotes to eukaryotes. It is published publishes results of a wide variety of studies from human and from informative model Physiological Genomics
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