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Interleukin-6 and tumor necrosis factor-α levels increase in response to maximal exercise in patients with chronic heart failure
Abstract Chronic heart failure (CHF) is characterized by the activation of neurohormones and cytokines. Strenuous exercise causes activation of both systems but the effect of acute bouts of exercise… Expand
Proinflammatory cytokine activation is linked to apoptotic mediator, soluble Fas level in patients with chronic heart failure.
The Fas/Fas Ligand system is a major apoptosis signaling pathway that is up-regulated in patients with chronic heart failure (CHF). Serum soluble Fas (sFas) levels increase in proportion to the CHF… Expand
A case of atrial tachycardia sensitive to increased caffeine intake.
A 33-year-old Japanese man with atrial tachycardia visited our clinic. He regularly consumed daily alcohol with cola, one cup of regular coffee, and a candy containing 0.7 mg of caffeine per tablet.… Expand
Uric Acid-Lowering Treatment With Benzbromarone in Patients With Heart FailureCLINICAL PERSPECTIVE
Background— Hyperuricemia is common in chronic heart failure (CHF), and it is a strong independent marker of prognosis. Upregulated xanthine oxidase (XO) activity and impaired renal excretion have… Expand
Impairment of urate excretion is an important mechanism for hyperuricemia in patients with heart failure
QRST integral analysis of body surface electrocardiographic mapping for assessing exercise-induced changes in the spatial distribution of local repolarization properties in patients with coronary…
Abstract We studied resting, postexercise, difference (postexercise - rest) QRST isointegral maps, and the correlation coefficient between resting and postexercise maps. Study I Fifteen controls and… Expand
Augmented response of plasma brain natriuretic peptide during exercise in patients with left ventricular dysfunction and heart failure
IMPAIRED VAGAL CONTROL OF HEART RATE IN DOGS WITH LEFT VENTRICULAR DYSFUNCTION WITHOUT HEART FAILURE
P508 CARDIAC AND PLASMA CATECHOLAMINE RESPONSES TO EXERCISE IN RELATION TO CARDIO-CEREBROVASCULAR EVENTS IN DIABETES MELLITUS