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C5a controls TLR-induced IL-10 and IL-12 production independent of phosphoinositide 3-kinase.
- Natsumi Okazaki, K. Hazeki, T. Izumi, K. Nigorikawa, O. Hazeki
- Biology, Medicine
- Journal of biochemistry
- 1 March 2011
The complement system is a classic central player in innate immunity. Most pathogens activate both complement and the toll-like receptor (TLR) pathway. Therefore, to provide a more comprehensive… Expand
Myeloproliferative leukemia protein activation directly induces fibrocyte differentiation to cause myelofibrosis
Myelofibrosis (MF) may be caused by various pathogenic mechanisms such as elevation in circulating cytokine levels, cellular interactions and genetic mutations. However, the underlying mechanism of… Expand