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Maximal and submaximal forces of slow fibers in human soleus after bed rest.
TLDR
The results indicate that long-term bed rest results in reductions of fiber size, force-generation capacity, and Ca(2+) sensitivity, and enhancement of shortening velocity in slow fibers of the soleus. Expand
Myonuclear domain and myosin phenotype in human soleus after bed rest with or without loading.
TLDR
It is shown that a single daily 10-h bout of modest muscle loading can prevent bed-rest-induced soleus fiber atrophy but has minimal effect on myosin phenotype, indicating that the specific adaptive cellular strategies involved may be a function of the duration and magnitude of the adaptive stimulus as well as the immediate activity history of the fiber before the new functional demands. Expand
Responses of muscle mass, strength and gene transcripts to long-term heat stress in healthy human subjects
TLDR
The results suggested that long-term application of heat stress could be effective in increasing the muscle strength associated with hypertrophy without exercise training. Expand
Effects of heat stress and mechanical stretch on protein expression in cultured skeletal muscle cells
TLDR
Observations suggest strongly that heating and passive stretch of muscle may be useful as a means of increasing muscle mass, not only in athletes but also in patients during rehabilitation. Expand
Elastic filaments in situ in cardiac muscle: deep-etch replica analysis in combination with selective removal of actin and myosin filaments
TLDR
This work selectively removed actin and myosin filaments from cardiac muscle fibers by gelsolin and potassium acetate treatment and observed the residual elastic filament network by deep-etch replica electron microscopy, leading to the conclusion that, in intact sarcomeres, the elastic filaments were laterally associated with myosIn and I bands, respectively. Expand
Changes in PKB/Akt and calcineurin signaling during recovery in atrophied soleus muscle induced by unloading.
TLDR
Findings are consistent with the hypothesis that PKB/Akt and its downstream mediators are active in the regrowth of muscle mass during the early periods of recovery from muscle atrophy and support the concept that CaN is involved in muscle remodeling during the later phases of Recovery from disuse Muscle atrophy. Expand
Involvement of AMPK in regulating slow-twitch muscle atrophy during hindlimb unloading in mice.
TLDR
The results imply that AMPK partially regulates unloading-induced atrophy of slow-twitch muscle possibly through modulation of the protein degradation system, especially the ubiquitin-proteasome system. Expand
Calcium and magnesium contents and volume of the terminal cisternae in caffeine-treated skeletal muscle
TLDR
The sustained Ca release caused by caffeine in situ contrasts with the transient Ca release observed in studies of fragmented SR preparations, and could be explained by mediation of the caffeine-induced Ca release by a second messenger produced more readily in intact muscle than in isolated SR. Expand
Administration of granulocyte colony-stimulating factor facilitates the regenerative process of injured mice skeletal muscle via the activation of Akt/GSK3αβ signals
TLDR
It was suggested that G-CSF treatment may facilitate the regeneration of injured skeletal muscles via the activation of Akt/GSK3αβ signals. Expand
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