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Lead and other trace metals in preeclampsia: a case-control study in Tehran, Iran.
TLDR
Environmental exposure to Pb, Sb and Mn may increase the risk of preeclampsia in women without occupational exposure; levels of metals in UCB to be sensitive indicators of female reproductive toxicity as compared with those in mother MWB. Expand
Assessment of Urinary Cotinine as a Marker of Nicotine Absorption from Tobacco Leaves: A Study on Tobacco Farmers in Malaysia
TLDR
As tobacco farmers evidence a risk of nicotine poisoning from tobacco leaves, assessment including GTS together with effects of pesticides will be necessary. Expand
Biomarkers of lead exposure.
  • T. Sakai
  • Chemistry, Medicine
  • Industrial health
  • 1 April 2000
TLDR
Of the biomarkers of lead exposure, blood lead (Pb-B), mainly red cell lead, is a representative of soft tissue lead, and most widely used as measures of body burden and absorbed (internal) doses of lead. Expand
Determination of urinary alkoxyacetic acids by a rapid and simple method for biological monitoring of workers exposed to glycol ethers and their acetates
TLDR
The present results indicate that determination of urinary alkoxyacetic acids by the practical method used here is useful for evaluating excessive exposure to solvents. Expand
Biological monitoring of workers exposed toN,N-dimethylformamide by determination of the urinary metabolites,N-methylformamide andN-acetyl-S-(N-methylcarbamoyl) cysteine
Biological monitoring of workers exposed toN,N-dimethylformamide (DMF) was carried out by determination of the urinary metabolites,N-methylformamide (MF, mainly fromN-hydroxymethylformamide)Expand
Gaschromatographic determination of butoxyacetic acid after hydrolysis of conjugated metabolites in urine from workers exposed to 2-butoxyethanol
TLDR
The determination of total BAA in urine is a suitable test for the biological monitoring of BE exposure, because of the simplicity in procedures and the good agreement with BE exposure levels. Expand
Relationships between distribution of lead in erythrocytes in vivo and in vitro and inhibition of ALA-D.
TLDR
Of three treatments or agents recovering the enzyme from lead effects, zinc is the only one that can fully restore the inhibition and lead responsible for inducing the inhibition is not bound to haemoglobin fraction proteins but to ALA-D fraction proteins. Expand
delta-Aminolevulinic acid in plasma or whole blood as a sensitive indicator of lead effects, and its relation to the other heme-related parameters.
TLDR
ALA-P is the best discriminators of lead exposure form baseline to high levels of exposure and ALA-U and zinc protoporphyrin were less effective for the diagnosis of lead Exposure than ALAD and ALa-P. Expand
δ-Aminolevulinic acid in plasma or whole blood as a sensitive indicator of lead effects, and its relation to the other home-related parameters
To evaluate the subclinical effect of lead exposure, we determined δ-aminolevulinic acid (ALA) levels in plasma (ALA-P), blood (ALA-B), and urine (ALA-U) and the activity of δ-aminolevulinic acidExpand
Relationship between delta-aminolevulinic acid dehydratase genotypes and heme precursors in lead workers.
TLDR
ALAD1 homozygotes might be more susceptible than ALAD2 carriers to disturbances in heme metabolism caused by lead exposure, and they showed significantly higher levels of ZP and ALA-P in comparison with those of ALad2 carriers at Pb-B levels more than 20 microg/dL and 40 microg/. Expand
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