T. Vinod Kumar Gupta

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Motion sickness is a common and distressing but poorly understood syndrome associated with nausea/vomiting and autonomic nervous system accompaniments that develops in the air or space as well as on sea or land. A bidirectional aetiologic link prevails between migraine and motion-sickness. Motion sickness provokes jerk nystagmus induced by both optokinetic(More)
In this paper, we present a novel methodology for processor evaluation in an embedded systems design environment. This evaluation can help in either selecting a suitable processor core or in evaluating changes to an ASIP. The processor evaluation is carried out in two stages. First, an architecture independent stage in which processors are rejected based on(More)
PVC based membranes of disodium salt of porphyrin 3,7,12,17-tetramethyl-8, 13-divinyl 2,18-porphine dipropionic acid (I) as ionophore with sodium tetra phenyl borate (NaTPB) as anion excluder and dibutyl phthalate (DBP), dioctyl phthalate (DOP), dibutyl butyl phosphonate (DBBP), tris(2-ethyl hexyl)phosphate (TEP), tri-n-butylphosphate (TBP) and(More)
Cortical spreading depression (CSD) has been at the center stage of migraine pathophysiology for approximately six decades. Reanalysis of CSD reveals several major unbridgeable gaps in this experimental neurophysiologic concept for migraine. Key phenotypic and pharmacological features of migraine challenge the assumed pathophysiologic role of CSD. Detection(More)
Migraine has been associated with patent foramen ovale (PFO), and PFO closure has become the most high-profile nonpharmacologic invasive therapy recommended for the prevention of recurrent migraine attacks, as well as for preventing further attacks in cryptogenic stroke. The results of Migraine Intervention with STARFlex Technology (MIST), a controversial(More)
Coppola et al., (2005) studied somatosensory evoked high-frequency oscillations (HFO) in migraine patients between attacks and advances the hypothesis that the habituation deficit in migraine patients is owing to a reduced pre-activation level of sensory cortices. While further proposing that a reduced interictal excitation ('pre-activation') level might be(More)