Susanne Wydenkeller

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A large percentage of spinal cord-injured subjects suffer from neuropathic pain below the level of the lesion (bNP). The neural mechanisms underlying this condition are not clear. The aim of this study was to elucidate the general effects of spinal deafferentiation and of bNP on electroencephalographic (EEG) activity. In addition, the relationship between(More)
Patients with incomplete spinal cord injury (iSCI) have impaired postural control leading to a high danger of falling. Clinically, it is impossible to assess the extent to which postural instability due to sensorimotor deficit is influenced by a disturbance in the vestibulospinal pathways. Galvanic vestibular stimulation (GVS) was applied to investigate(More)
OBJECTIVE Laser-evoked potentials have been shown to be clinically useful for the electrophysiological assessment of nociceptive pathways. Contact heat evoked potentials (CHEP) are less established but might be advantageous for clinical purposes. This study aimed at determining the conduction velocity (CV) of central pain (spinothalamic tract, STT) pathways(More)
Spinothalamic tract (STT) dysfunction seems to be crucially involved in the development of central neuropathic pain (NP) after spinal cord injury (SCI). However, previous attempts to identify differences in the extent or location of STT damage between subjects with and without NP failed. Here we show that the spontaneous recovery of human STT function(More)
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