Sumito Teshima

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The phenotypes of mice lacking peptidyl prolyl cis/trans isomerase Pin1 (Pin1(-/-)) indicated that deficient Pin1 might be related to a variety of diseases. We created TAT-Pin1, a fusion protein of human immunodeficiency virus 1 trans-activator of transcription factor with Pin1. Treatment of HeLa cells with TAT-Pin1 increased the ratio of the S phase.(More)
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