Steven Armentrout

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Biological experience and intuition suggest that self-replication is an inherently complex phenomenon, and early cellular automata models support that conception. More recently, simpler computational models of self-directed replication called sheathed loops have been developed. It is shown here that "unsheathing" these structures and altering certain(More)
We examine opportunistic evolution, a variation of master-slave distributed evaluation designed for deployment of evolutionary computation to very large grid computing architectures with limited communications, severe evaluation overhead, and wide variance in evaluation node speed. In opportunistic evolution, slaves receive some N individuals at a time,(More)
Chloramphenicol can inhibit protein synthesis in mammalian cell-free systems as effectively as it inhibits protein synthesis in analogous microbial systems. Significant inhibition in mammalian systems was obtained only when protein synthesis was stimulated by the addition of template RNA to the system, there being comparatively little inhibition of protein(More)
Replica exchange molecular dynamics (REMD) has become a valuable tool in studying complex biomolecular systems. However, its application on distributed computing grids is limited by the heterogeneity of this environment. In this study, we propose a REMD implementation referred to as greedy REMD (gREMD) suitable for computations on heterogeneous grids. To(More)
The workshop on petroleum applications of EC, new Rubik's cube competition, best paper nominees The Columns competitions @ WCCI2008 forthcoming papers calls & calendar EDITORIAL Editorial S pace Invaders was the first video game I ever played. There were invaders from outer space and I defended the Earth with a small cannon and the little cover provided by(More)
Large-scale genetic studies of common human diseases have focused almost exclusively on the independent main effects of single-nucleotide polymorphisms (SNPs) on disease susceptibility. These studies have had some success, but much of the genetic architecture of common disease remains unexplained. Attention is now turning to detecting SNPs that impact(More)
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