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  • Valeria Bafunno, Davide Firinu, +9 authors Maurizio Margaglione
  • Medicine
  • The Journal of allergy and clinical immunology
  • 2018 (First Publication: 8 June 2017)
  • Background: Hereditary angioedema (HAE) is a rare genetic disease usually caused by mutation in the C1 inhibitor or the coagulation Factor XII gene. However, in a series of patients with HAE, noContinue Reading
  • Francescopaolo Granata, Annunziata Frattini, +8 authors Massimo Triggiani
  • Biology, Medicine
  • Journal of immunology
  • 2010 (First Publication: 1 May 2010)
  • Angiogenesis and lymphangiogenesis mediated by vascular endothelial growth factors (VEGFs) are main features of chronic inflammation and tumors. Secreted phospholipases A(2) (sPLA(2)s) areContinue Reading
  • Massimo Triggiani, Angelica Petraroli, +7 authors Giancarlo Marone
  • Biology, Medicine
  • The Journal of allergy and clinical immunology
  • 2007 (First Publication: 1 February 2007)
  • BACKGROUND Histamine modulates several functions in human monocytes, macrophages, and dendritic cells. However, responses elicited by histamine differ depending on cell type, suggesting variableContinue Reading
  • Rosaria Ilaria Staiano, Stefania Loffredo, +12 authors Giancarlo Marone
  • Biology, Medicine
  • Journal of leukocyte biology
  • 2016 (First Publication: 1 April 2016)
  • Macrophages are pivotal effector cells in immune responses and tissue remodeling by producing a wide spectrum of mediators, including angiogenic and lymphangiogenic factors. Activation of cannabinoidContinue Reading
  • Francescopaolo Granata, Annunziata Frattini, +4 authors Massimo Triggiani
  • Biology, Medicine
  • European journal of immunology
  • 2006 (First Publication: 1 July 2006)
  • Secretory phospholipases A(2) (sPLA(2)) are enzymes released during inflammatory reactions. These molecules activate immune cells by mechanisms either related or unrelated to their enzymaticContinue Reading
  • Valeria Bafunno, Maria Bova, +6 authors Massimo Triggiani
  • Medicine, Biology
  • Annals of human genetics
  • 2014 (First Publication: 1 March 2014)
  • Hereditary angioedema (HAE) is an autosomal dominant disease due to mutations in the C1 inhibitor gene (C1NH) that affects protein synthesis (HAE type I) or function (HAE type II). In 45 subjectsContinue Reading