Shoji Miyaguchi

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1 FEAL cipher family FEAL-8 has been expanded to FEAL-N (N round FEAL with 64-bit key), where FEAL-N with N=4/8 is identical to FEAL-4/8 which have been previously published respectively. N is a user defined parameter (N>4, N:even, N=2”, 2 >2 is recommended). FEAL-N has also been expanded to FEAL-NX (X: expansion, N round FEAL with 12%bit key) that accepts(More)
ESIGN is an efficient digital signature algorithm [OkS, Ok], whose computation speed is more than twenty times faster than that of the RSA scheme, while its key length and signature length are comparable to those of the M A scheme. This paper presents a software implementation of ESIGN on an 8bit micro-processor smart card. This realizes a computation time(More)
N-acetylcysteine and pentoxifylline, free radical scavengers and inhibitors of tumor necrosis factor-alpha (TNF-alpha) production, inhibit the development of peripheral neuropathy in streptozotocin (STZ)-induced diabetic rats. This study was designed to elucidate the effect of gliclazide, an oral hypoglycemic sulfonylurea, on diabetic neuropathy, because it(More)
Dr. Shamir demonstrated an attack method for FEAL-8 (eight round FEAL) and eight round DES at Securicom ‘89, this past March. Some technical Journals have reported that the attack method he demonstrated was type (l), but we feel that the attack was actually type (3). It appears to us that Shamir’s method estimates the key using only the ciphertexts that are(More)
It is well known that some anti-hypertensive drugs affect insulin sensitivity and that tumor necrosis factor-alpha (TNF-alpha) is a mediator of obesity-associated insulin resistance. In this study, we have investigated the effect of anti-hypertensive drugs, calcium (Ca) channel blockers (amlodipine, manidipine and nicardipine), an alpha(1)-blocker(More)
Nicotinamide, a pellagra-preventive factor, has multiple functions such as inhibition of poly-ADP-ribose synthetase, inhibition of inducible nitric oxide synthase, free radical scavenging and suppression of major histocompatibility complex class II expression and ICAM-1 expression on endothelial cells. In addition to these, we have found an inhibitory(More)
It has been reported that angiotensin converting enzyme (ACE) inhibitors have beneficial effects on insulin resistance and congestive heart failure, in which elevations of serum tumor necrosis factor-alpha (TNF-alpha) level have been indicated. Therefore, in this study, we examined effect of ACE inhibitors on TNF-alpha production both in vitro and in vivo(More)
It has recently been indicated that tumor necrosis factor-alpha (TNF-alpha) production is increased under chronic hyperglycemia and TNF-alpha has harmful effects on insulin sensitivity and possibly on chronic diabetic complications. Therefore it will be favorable for diabetes treatment if anti-diabetic agents also have anti-TNF-alpha activities. In this(More)