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Cutaneous leishmaniasis (CL) which caused by the different species of Leishmaina, produces a skin ulcer that heals spontaneously in most cases, leaving an unsightly scar. Effort to develop an effective vaccine so far had been limited due to lack of an appropriate adjuvant Protection against leishmaniasis depends on induction of T h1 response or cell(More)
The profile of central (=TCM) and effector (=TEM) memory CD4+ T cell subsets and the possible role as surrogate markers of protection is studied in the volunteers with history of cutaneous leishmaniasis (HCL). Profile of T cell subsets based on CCR7/CD45RA expressions and phenotypic changes after soluble Leishmania antigen (SLA) stimulation were analyzed.(More)
The inoculation of live Leishmania (L.) major to produce a single lesion is called leishmanization (LZ). LZ lesion upon cure prevents further natural infection which might be multiple lesions on unwanted sites such as face. Cutaneous leishmaniasis (CL) usually leads to a self healing lesion; though rarely the lesion persists and becomes refractory to all(More)
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