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Watzka et al. [1] have recently reported the phenotype of patients with therapeutic resistance to 3-substituted-4-hydroxycoumarin oral anticoagulants (OACs) and with point mutations inVKORC1, which(More)
Acute promyelocytic leukaemia (APL) is characterized by the t(15;17) leading to the formation of PML-RARalpha and RARalpha-PML fusion genes; this rearrangement has been considered both diagnostic(More)
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