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  • P Y Perera, Tanya N. Mayadas, +4 authors Stefanie N Vogel
  • Medicine, Biology
  • The Journal of Immunology
  • 2001 (First Publication: 1 January 2001)
  • Overproduction of inflammatory mediators by macrophages in response to Gram-negative LPS has been implicated in septic shock. Recent reports indicate that three membrane-associated proteins, CD14,Expand
  • Norbert Reiling, Christoph Hoelscher, +4 authors Stefan Ehlers
  • Biology, Medicine
  • The Journal of Immunology
  • 2002 (First Publication: 1 October 2002)
  • Innate resistance against Mycobacterium tuberculosis is thought to depend critically on engagement of pattern recognition receptors on macrophages. However, the relative contribution of theseExpand
  • Alain Haziot, S. Chen, Enza Ferrero, Martin G. Low, Robert Silber, Sanna M. Goyert
  • Biology, Medicine
  • Journal of immunology
  • 1988 (First Publication: 15 July 1988)
  • CD14 is a myeloid differentiation Ag expressed primarily on peripheral blood monocytes and macrophages. Although its function is unknown, the CD14 gene maps to a region encoding several myeloidExpand
  • Timothy J. Sellati, Deborah A. Bouis, +7 authors Justin D. Radolf
  • Biology, Medicine
  • Journal of immunology
  • 1998 (First Publication: 1 June 1998)
  • Lipoproteins of Treponema pallidum and Borrelia burgdorferi possess potent proinflammatory properties and, thus, have been implicated as major proinflammatory agonists in syphilis and Lyme disease.Expand
  • P Y Perera, Stefanie N Vogel, G R Detore, Alain Haziot, Sanna M. Goyert
  • Biology, Medicine
  • Journal of immunology
  • 1997 (First Publication: 1 May 1997)
  • The antitumor agent, Taxol, shares with bacterial LPS the ability to activate murine macrophages, and its LPS-mimetic effects are blocked by LPS analogue antagonists. Since CD14 is central to theExpand
  • Graciela Andonegui, Sanna M. Goyert, Paul Kubes
  • Medicine, Biology
  • The Journal of Immunology
  • 2002 (First Publication: 15 August 2002)
  • The objective of this study was to systematically assess leukocyte-endothelial cell interactions in vivo in response to LPS in CD14-deficient (CD14−/−) and Toll-like receptor 4-deficient (TLR4d;Expand