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Unliganded Thyroid Hormone Receptor- 1 Represses Liver X Receptor /Oxysterol-Dependent Transactivation
It is suggested that T3 and TR influence lipid metabolism regulated by oxysterol/LXR at the transcriptional level, and the interaction between TR 1 and corepressors is important to the unliganded TR-mediated suppression of LXR -transactivation.
Unliganded thyroid hormone receptor-beta1 represses liver X receptor alpha/oxysterol-dependent transactivation.
It is suggested that T3 and TR influence lipid metabolism regulated by oxysterol/LXRalpha at the transcriptional level, and the interaction between TRbeta1 and corepressors is important to the unliganded TR-mediated suppression of LXRalpha-transactivation.
The role of the amino-terminal domain in the interaction of unliganded peroxisome proliferator-activated receptor gamma-2 with nuclear receptor co-repressor.
- Sadako Suzuki, S. Sasaki, +6 authors Hirotoshi Nakamura
- Biology, MedicineJournal of molecular endocrinology
- 1 September 2010
Inter-domain communication in PPARG2 regulates not only lig and-dependent transactivation but also ligand-independent silencing, which is essential for total elimination of the influence of ligands.
Influence of thyroid dysfunction on brain natriuretic peptide level in health examination participants.
A direct stimulatory effect of thyroid hormone on the secretion (or production) of BNP was confirmed even in a large number of health examination participants.
Interview with Sadako Suzuki
A woman, orphaned at an early age, remembers her parents, adoption by a Waiakea family, childhood jobs, and schooling. She relates the background of Hilo Products and her marriage to a son of the…
MON-619 The Direct Stimulatory Effect of Thyroid Hormone Against Plasma Brain Natriuretic Peptide in Health Check-Up Participants: Cross-Sectional and Longitudinal Studies
The study suggests that plasma BNP concentrations are not sufficiently sensitive to evaluate TH activity in the heart, and Physicians should consider multiple independent contributing factors other than thyroid dysfunction when interpreting BNP data in patients with thyroid disorders.