Sabine Bauer

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  • Luisa Schmidt, Marc Wiedner, Georgia Velimezi, Jana Prochazkova, Michel Owusu, Sabine Bauer +1 other
  • 2014
Unresolved replication intermediates can block the progression of replication forks and become converted into DNA lesions, hence exacerbating genomic instability. The p53-binding protein 1 (53BP1) forms nuclear bodies at sites of unrepaired DNA lesions to shield these regions against erosion, in a manner dependent on the DNA damage kinase ATM. The molecular(More)
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