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Induction of suppressor T cells by interleukin 2.
TLDR
It was shown that the addition of high concentrations of IL 2 suppressed the generation of alloreactive CTL in conventional MLC, indicating that IL 2 may play a dual role in the regulation of CTL responses.
Analysis of High-Molecular-Weight Ribonucleic Acid Associated with Intracisternal A Particles
TLDR
There are probably some taxonomic relationships between intracisternal A particles and oncogenic RNA viruses, and this HMW RNA is sensitive to ribonuclease digestion and alkali treatment but is resistant to Pronase treatment.
FMNL2 destabilises COMMD10 to activate NF-κB pathway in invasion and metastasis of colorectal cancer
TLDR
These data demonstrate that the FMNL2/COMMD10/p65 axis acts as a critical regulator in the maintenance of metastatic phenotypes and is strongly associated with negative clinical outcomes.
Dose dependency of aflatoxin B1 binding on human high molecular weight DNA in the activation of proto-oncogene.
TLDR
Excessive AFB1 binding on the hHC and PLC HMW DNAs resulted in an "over-kill" of both cell transformation capability and templating activity of the DNA.
Abrogation of cell‐mediated immunity by hyperimmune alloantiserum: Mechanisms and correlation with allograft enhancement
Alloantiserum blocking of cell‐mediated immunity as measured by a 51chromium release cell‐mediated cytotoxicity assay has been characterized and correlated with immunological enhancement of tumor
Characterization of an Endogenous RNA-Dependent DNA Polymerase Associated with Murine Intracisternal A Particles
TLDR
Sensitivity of the endogenous RNA-dependent DNA polymerase activity to a low concentration of pancreatic ribonuclease in the presence of a high concentration of NaCl suggested that the enzyme might be utilizing the A particle endogenous RNA as template.
Lymphokine-induced cytotoxicity: characterization of effectors, precursors, and regulatory ancillary cells.
TLDR
The serological phenotype of lymphokine-induced cytotoxic cell effectors were found to be Thy 1+, Lyt 2-, and AGM1-; therefore, they were neither classic natural killer (NK) cells nor cytot toxic T-lymphocytes.
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