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The effects of total lymphocyte deficiency on the extent of atherosclerosis in apolipoprotein E-/- mice.
Activated T lymphocytes are present in human atherosclerotic lesions and autoantibodies to antigens within lesions have been detected in serum, but the roles of the cellular and humoral immuneExpand
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Lymphocyte populations in atherosclerotic lesions of apoE -/- and LDL receptor -/- mice. Decreasing density with disease progression.
Lymphocytes are prominent components of human atherosclerotic lesions, but their presence in murine models of disease has not been confirmed. Lymphocyte subpopulations have been identified in apoEExpand
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Apolipoprotein E-deficient mice have impaired innate immune responses to Listeria monocytogenes in vivo.
Apolipoprotein E (apoE) influences both innate and acquired immunity in cultured cells. To determine whether apoE affects the immune system in vivo, Listeria monocytogenes (LM) was administeredExpand
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Evidence for non-uniform distribution of D-glucose within human red cells during net exit and counterflow.
The kinetic parameters of net exit of D-glucose from human red blood cells have been measured after the cells were loaded to 18 mM, 75 mM and 120 mM at 2 degrees C and 75 mM and 120 mM at 20 degreesExpand
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Enhanced development of atherosclerosis in cholesterol-fed rabbits by suppression of cell-mediated immunity.
T lymphocytes are present in atherosclerotic lesions, but the role of this cell type in the disease process has not been determined. To determine whether cell-mediated immunity influencesExpand
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Selegiline in narcolepsy.
We examined the effect of the specific monoamine oxidase-B (MAO-B) inhibitor selegiline (deprenyl, Eldepryl), 20-30 mg p.o. daily, in 21 subjects with the narcoleptic syndrome for 4 weeks. SelegilineExpand
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Lipopolysaccharide decreases scavenger receptor mRNA in vivo.
Lipopolysaccharide (LPS) downregulates scavenger receptor (ScR) activity in cultured macrophages through release of tumor necrosis factor-alpha (TNF-alpha). However, LPS administration in vivoExpand
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