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Effect of salivary epidermal growth factor on wound healing of tongue in mice.
TLDR
The results suggest that salivary EGF is involved in the promotion of wound healing of the tongue in both male and female mice. Expand
The role of transcription and messenger RNA stability in the regulation of epidermal growth factor receptor gene expression in regenerating mouse liver
TLDR
The results suggest that transcription induced by partial hepatectomy requires protein synthesis and that labile proteins are involved in the regulation of the stability of epidermal growth factor receptor messenger RNA. Expand
Influence of epidermal growth factor on liver regeneration after partial hepatectomy in mice.
TLDR
The role of epidermal growth factor (EGF) in liver regeneration was studied in mice after partial hepatectomy and treatment of sialoadenectomized mice with EGF completely restored the pattern of DNA synthesis so that a sharp peak appeared at 48 h. Expand
Pretranslational enhancement of epidermal growth factor receptor by direct effect of testosterone in mouse liver.
TLDR
Results suggest that hepatic EGFR levels are at least in part regulated at a pretranslational level by direct effects of androgens on the liver, and that endogenous EGF is not involved in the androgenic regulation of hepatics. Expand
Growth control of androgen-dependent Shionogi carcinoma cells by growth factor(s) produced from androgen-independent Shionogi carcinoma cells in a paracrine mechanism.
TLDR
The results suggest that the growth of androgens-dependent SC3 cells is stimulated by androgen-induced growth factor(s) produced from the same cells (autocrine mechanism) and is also regulated by autonomous growth factor produced from androgen -independent cancer cells formed from the dependent cancer cells (paracrine mechanism). Expand
Stimulative effect of physiological doses of androgen or pharmacological doses of estrogen on growth of Shionogi carcinoma 115 in mice.
TLDR
It is concluded that high doses of 17 beta-estradiol, which binds to androgens receptor with relatively low but significant affinity, enhance the tumor growth not via the androgen receptor system. Expand
Growth-stimulative effect of estrogen on androgen-dependent Shionogi carcinoma 115.
TLDR
The results demonstrate for the first time the stimulative effect of estrogen on the growth of androgen-dependent Shionogi carcinoma 115, which contains typical estrogen receptor, which might be able to transmit estrogen signal to tumor cell nuclei with regard to tumor growth. Expand
Growth-stimulating effect of pharmacological doses of estrogen on androgen-dependent Shionogi carcinoma 115 in vivo but not in cell culture.
TLDR
The growth of SC115 cells is stimulated by either physiological doses of androgen or pharmacological doses of estrogen in vivo but only by androgen in cell culture. Expand
Growth-stimulatory effect of androgen-induced autocrine growth factor(s) secreted from Shionogi carcinoma 115 cells on androgen-unresponsive cancer cells in a paracrine mechanism.
TLDR
The present findings demonstrate that T-induced autocrine GF-high secreted from SC-3 cells can also stimulate the growth of progressed unresponsive SC-4 cells in a paracrine mechanism and that autonomous GF-low secreting from both SC-1 and SC-2 cells can stimulate thegrowth of only SC- 4 cells. Expand
Action mechanisms of physiological doses of androgen or pharmacological doses of estrogen in growth stimulation of Shionogi carcinoma 115 in mice.
TLDR
The present findings suggest that growth-stimulatory activities of androgen and high doses of estrogen onSC115 cells are mediated by growth factor(s), secreted from SC115 cells through androgen receptor and from some of nontransformed cells through estrogen receptor, respectively. Expand
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