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Changing internal constraints on action: the role of backward inhibition.
The authors tested the hypothesis that disengagement during intentional shifts between task sets is accompanied by inhibition of the previous task set ("backward inhibition") and predicted increased response times when shifting to a task set that had to be abandoned recently and, thus, suffers residual inhibition.
On the genesis of abstract ideas.
Timing Functions of The Cerebellum
The results suggest that the domain of the cerebellar timing process is not limited to the motor system, but is employed by other perceptual and cognitive systems when temporally predictive computations are needed.
Attentional and Nonattentional Forms of Sequence Learning
The results suggest that attentional and nonattentional learning operate independently, in parallel, do not share information, and represent sequential information in qualitatively different ways.
The cognitive and neural architecture of sequence representation.
- S. Keele, R. Ivry, U. Mayr, E. Hazeltine, H. Heuer
- Psychology, BiologyPsychological review
- 1 April 2003
The authors theorize that 2 neurocognitive sequence-learning systems can be distinguished in serial reaction time experiments, one dorsal (parietal and supplementary motor cortex) and the other ventral (temporal and lateral prefrontal cortex), which are relevant to issues of attentional effects on learning.
Attention and structure in sequence learning.
In this study, the role of attention, sequence structure, and effector specificity in learning a structured sequence of actions is investigated.
Processing of visual feedback in rapid movements.
Dissociation of the lateral and medial cerebellum in movement timing and movement execution
It is concluded that the lateral regions of the cerebellum are critical for the accurate functioning of an internal timing system.
Toward a Functional Analysis of the Basal Ganglia
- A. Hayes, M. Davidson, S. Keele, R. Rafal
- Psychology, BiologyJournal of Cognitive Neuroscience
- 1 March 1998
Using the framework of equilibrium point theory of movement, it is discussed how a set switching deficit may also underlie clinical motor disturbances seen in Parkinson's disease.