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Variable clinical presentation in three patients with 3-methylglutaconyl-coenzyme A hydratase deficiency
TLDR
3 patients with 3- MG-CoA hydratase deficiency who also manifested widely differing clinical presentations are presented, bringing to eight the total number of patients with documented 3-MG-Co A hydr atase deficiency.
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The vascular bed as the primary target in the destruction of skin grafts by antiserum. I. Resistance of freshly placed xenografts of skin to antiserum
TLDR
It is shown that the initial period of resistance to antiserum is due to factors acting locally within the graft and is entirely uninfluenced by the regimen of immunosuppression or the protective dressings that are used, and supports the idea that the induction of vascular injury is an essential step in antisera-mediated damage to tissue grafts.
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The vascular bed as the primary target in the destruction of skin grafts by antiserum. II. Loss of sensitivity to antiserum in long-term xenografts of skin
TLDR
It was found that the acquisition of resistance is correlated with replacement of graft endothelium by host cells, as demonstrated by the use of immunofluorescent techniques in conjunction with mouse anti-rat serum and rat anti-mouse serum.
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The detection of 3-methylglutarylcarnitine and a new dicarboxylic conjugate, 3-methylglutaconylcarnitine, in 3-methylglutaconic aciduria.
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Acute destruction of rat skin grafts by alloantisera.
TLDR
Rat skin transplanted to mice or to allogeneic rats can be acutely and severely damaged by injecting the graft recipients with alloimmune serum of appropriate specificity, and combinations of the two sera can act synergistically to destroy LBN skin that has been grafted to immunosuppressed mice.
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Strain variations in the responses of mice to xenografts of skin.
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Variations in the responses of mouse strains to rat xenografts.
TLDR
There is strong histoincompatibility between the rat and the mouse such that grafts of rat skin or primarily vascularized hearts placed in mice undergo acute rejection about 5 to 9 days after transplantation, and this phenomenon is not restricted to skin grafts.
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Destruction of rat skin grafts by humoral antibody.
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The manual tests that can elicit the strongest responses from the palmaris longus muscle
Changes in sensitivity to immune destruction of long-term skin grafts.
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