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Prenatal development of gabaergic, glycinergic, and dopaminergic neurons in the rabbit retina
TLDR
By the authors' criteria, putative GABAergic and glycinergic amacrine cells are mature between postnatal day 10 (P10) and P12, around the time when the eyes first open and direction-selective ganglion cells can first be recorded. Expand
Postnatal development of dopaminergic neurons in the rabbit retina
TLDR
It is shown that certain neurons in the newborn retina already possess a specific mechanism for dopamine uptake, and the results indicate that, in the rabbit retina, the commitment for certain neurons to be dopaminergic is made prenatally. Expand
Postnatal development of GABA‐ergic neurons in the rabbit retina
TLDR
It is shown that certain neurons in the newborn retina already possess a specific mechanism for GABA uptake, and the commitment by certain neurons to use GABA as the transmitter is made prenatally. Expand
The role of pre-synaptic GABA and benzodiazepine receptors in the control of noradrenaline release in rat hippocampus
TLDR
Noradrenaline release from synaptosomes of rat hippocampus is modulated by both GABA and benzodiazepines, and agonists at GABAA and GABAB receptors enhance spontaneous release and depress K+ -evoked release, respectively. Expand
Postnatal development of glycinergic neurons in the rabbit retina
TLDR
The uptake and K+‐stimulated release of glycine are used as physiological probes to follow the emergence and maturation of putative glycinergic neurons during postnatal development of the rabbit retina and suggest that the glycine‐accumulating neurons may be a subpopulation of amacrine cells and may use glycine as a neurotransmitter. Expand
Effect of baclofen on in vitro noradrenaline release from rat hippocampus and cerebellum: an action at an α2-adrenoceptor
TLDR
It is concluded that baclofen is not a specific agonist at GABA(B) receptors in the brain. Expand
Studies on the mechanism of modulation of [3H]noradrenaline release from rat hippocampal synaptosomes by GABA and benzodiazepine receptors
TLDR
The results suggest that the GABA(A)-receptor mediated release of [(3)H]noradrenaline is due to depolarisation resulting from increased Cl(?) conductance whereas the depression of depolarised release by GABA(B) or benzodiazepine receptors is mediated by a cAMP-dependent decrease in the voltage-dependent Ca(2+) conductance. Expand
Effects of Guanyl Nucleotides on [3H]Flunitrazepam Binding to Rat Hippocampal Synaptic Membranes: Equilibrium Binding and Dissociation Kinetics
The effects of guanyl nucleotides on the binding of [3H]flunitrazepam to rat hippocampal synaptic membranes were studied. In equilibrium binding studies, γ‐amino‐n‐butyric acid (GABA) increased andExpand
Multiple effects of drugs acting on benzodiazepine receptors
TLDR
The results suggest that the distinction between agonist and inverse agonist applies to the GABA-dependent but not to the Benzodiazepine-independent action of benzodiazepines. Expand
Hiv-1 neutralizing monoclonal antibodies.
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