• Publications
  • Influence
Shiga-like toxin-converting phages from Escherichia coli strains that cause hemorrhagic colitis or infantile diarrhea.
Escherichia coli K-12 acquired the ability to produce a high titer of Shiga-like toxin after lysogenization by either of two different bacteriophages isolated from a highly toxinogenic EscherichiaExpand
Involvement of a plasmid in the invasive ability of Shigella flexneri.
Representative Shigella flexneri strains were studied to determine whether plasmids are involved in their virulence. All invasive S. flexneri strains, irrespective of serotype, were found to harbor aExpand
Pathogenesis of Escherichia coli diarrhea.
Abstract Two Escherichia coli strains isolated in Vietnam from American soldiers with diarrhea and acute "colitis" were examined for virulence in both in vitro and in vivo experimental models. Thei...
Production of Shigella dysenteriae type 1-like cytotoxin by Escherichia coli.
Strains of Escherichia coli previously implicated or proven to be causes of diarrhea were examined for production of a toxin similar to that of Shigella dysenteriae type 1 (Shiga). Organisms grown inExpand
Transmission of acute infectious nonbacterial gastroenteritis to volunteers by oral administration of stool filtrates.
Raphael Dolin, NeU R. Blacklow, Herbert DuPont, Samuel Formal, Robert F. Buscho, Julius A. Kasel, Robert P. Chames, Richard Hornick, and Robert M. Chanock From the Laboratories of Infectious DiseasesExpand
Pathogenesis of Shigella dysenteriae 1 (Shiga) dysentery.
Myron M. Levine, Herbert L. DuPont, Samuel B. Formal, Richard B. Hornick, Akio Takeuchi, Eugene J. Gangarosa, Merrill J. Snyder, and Joseph P. Libonati From the Shigella Vaccine Development Program,Expand
Inoculum size in shigellosis and implications for expected mode of transmission.
Pathogenesis of salmonellosis. Studies of fluid secretion, mucosal invasion, and morphologic reaction in the rabbit ileum.
Strains of Salmonella typhimurium were studied in the ligated rabbit ileal loop model to gain insight into the mechanisms whereby bacteria which invade the gastrointestinal mucosa evoke fluidExpand