Ryan L. Collins

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Users may view, print, copy, and download text and data-mine the content in such documents, for the purposes of academic research, subject always to the full Conditions of use: AUTHOR CONTRIBUTIONS Designed the study and wrote the manuscript (T.T., A.C.); edited manuscript (all authors); examined phenotype data for the female autism patients (T. SUMMARY(More)
Recurrent, reciprocal genomic disorders resulting from non-allelic homologous recombination (NAHR) between near-identical segmental duplications (SDs) are a major cause of human disease, often producing phenotypically distinct syndromes. The genomic architecture of flanking SDs presents a challenge for modeling these syndromes; however, the capability to(More)
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