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Reperfusion induces myocardial apoptotic cell death.

• Zhi-qing Zhao, Motoomi Nakamura, +5 authors Jakob Vinten-Johansen
• Cardiovascular research
• 2000

OBJECTIVE The purpose of the present study was to investigate whether apoptosis is triggered during ischemia (I) and reperfusion (R) and whether I/R-induced apoptosis is correlated with changes in… (More)

Off-pump multivessel coronary bypass via sternotomy is safe and effective.

• John D. Puskas, Connie E Wright, Russell S Ronson, W. M. Court Brown, Julie Gott, Robert A. Guyton
• The Annals of thoracic surgery
• 1998

BACKGROUND In an attempt to avoid the deleterious effects of cardiopulmonary bypass, off-pump coronary artery bypass grafting has been rediscovered and refined. The purpose of this study was to… (More)

Broad-spectrum cardioprotection with adenosine.

• Jakob Vinten-Johansen, Vinod H. Thourani, +5 authors Robert A. Guyton
• The Annals of thoracic surgery
• 1999

Ischemia-reperfusion results in contractile dysfunction, necrosis, and vascular injury. This postischemic injury is mediated in part by superoxide radical production, neutrophils, dysfunction to… (More)

The cardiovascular effects and implications of peroxynitrite.

• Russell S Ronson, Masafumi Nakamura, Jakob Vinten-Johansen
• Cardiovascular research
• 1999

Nitric oxide is an endogenous autacoid produced primarily by the vascular endothelium. Under basal conditions, nitric oxide undergoes a rapid biradical reaction with superoxide anions to form… (More)

Nonanticoagulant heparin inhibits NF-kappaB activation and attenuates myocardial reperfusion injury.

• Vinod H. Thourani, Somjot S. Brar, +7 authors Jakob Vinten-Johansen
• American journal of physiology. Heart and…
• 2000

Heparin reduces ischemia-reperfusion injury to myocardium. This effect has been attributed to complement inhibition, but heparin also has other activities that might diminish ischemia-reperfusion. To… (More)

Nitric oxide and the vascular endothelium in myocardial ischemia-reperfusion injury.

• Jakob Vinten-Johansen, Zuowei Zhao, +4 authors Robert A. Guyton
• Annals of the New York Academy of Sciences
• 1999

The normal coronary vascular endothelium (VE) tonically releases nitric oxide (NO) by converting L-arginine to citrulline by a constitutive NO synthase. Reperfusion after myocardial ischemia reduces… (More)

Glutathione reverses endothelial damage from peroxynitrite, the byproduct of nitric oxide degradation, in crystalloid cardioplegia.

• Masato Nakamura, Vinod H. Thourani, +9 authors Jakob Vinten-Johansen
• Circulation
• 2000

BACKGROUND NO has been advocated as an adjunct to cardioplegia solutions. However, NO undergoes a rapid biradical reaction with superoxide anions to produce peroxynitrite (ONOO(-)). ONOO(-) in… (More)

Peroxynitrite, the breakdown product of nitric oxide, is beneficial in blood cardioplegia but injurious in crystalloid cardioplegia.

• Russell S Ronson, Vinod H. Thourani, +6 authors Jakob Vinten-Johansen
• Circulation
• 1999

BACKGROUND Peroxynitrite (ONOO(-)) has been implicated as a primary mediator in the deleterious effects of nitric oxide (NO) in crystalloid solutions, possibly due to a lack of detoxification… (More)

Colonic necrosis subsequent to catheter-directed thrombin embolization of the inferior mesenteric artery via the superior mesenteric artery: a complication in the management of a type II endoleak.

• Ruth Lee Bush, Peter H Lin, Russell S Ronson, Brian S. Conklin, Louis G. Martin, Alan B. Lumsden
• Journal of vascular surgery
• 2001

The optimal management of endoleaks after endovascular repair of abdominal aortic aneurysms remains to be established. In this report, we describe a persistent side-branch, or type II, endoleak 1… (More)

Adenosine A3 pretreatment before cardioplegic arrest attenuates postischemic cardiac dysfunction.

• Vinod H. Thourani, Russell S Ronson, J. E. Jordan, Robert A. Guyton, Jakob Vinten-Johansen
• The Annals of thoracic surgery
• 1999

BACKGROUND The cardioprotective effects of the adenosine A3 receptor in a cardioplegia model have not been described. We tested the hypothesis that infusion of the A3 receptor agonist, Cl-IB-MECA… (More)

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