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Fetal Alcohol Exposure Alters Cerebrovascular Reactivity to Vasoactive Intestinal Peptide in Adult Sheep
Chronic fetal alcohol exposure impairs neural and vascular development. We have previously shown that fetal alcohol exposure is associated with attenuated hypoxic cerebral vasodilation and reduced… Expand
Effects of binge alcohol exposure in the second trimester on intracerebral arteriolar function in third trimester fetal sheep
Fetal alcohol syndrome is a leading cause of mental retardation, but mechanisms of alcohol-associated brain damage remain elusive. Chronic alcohol exposure attenuates fetal and neonatal hypoxic… Expand
Binge alcohol exposure in the second trimester attenuates fetal cerebral blood flow response to hypoxia.
- D. Mayock, Dana Ness, Robin L. Mondares, C. Gleason
- Journal of applied physiology
- 1 March 2007
Alcohol is detrimental to the developing brain and remains the leading cause of mental retardation in developed countries. The mechanism of alcohol brain damage remains elusive. Studies of… Expand
The effects of binge alcohol exposure in the 2nd trimester on the estimated density of cerebral microvessels in near-term fetal sheep
Heavy fetal alcohol exposure is associated with a spectrum of neurological abnormalities, although the mechanism of injury is largely unknown. We previously reported attenuated cerebral blood flow… Expand
The Effect of Binge Fetal Alcohol Exposure on the Number of Vasoactive Intestinal Peptide-Producing Neurons in Fetal Sheep Brain
Previously we demonstrated that fetal alcohol exposure attenuates hypoxic cerebral vasodilation in fetal and neonatal sheep. One mechanism may be altered expression of brain vasoactive substances. We… Expand