René Bindels

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Interleukin-18 (IL18) participates in atherogenesis through several putative mechanisms 1,2. Interruption of IL18 action reduces atherosclerosis in mice 3,4. This study shows that the absence of IL18 receptor (IL18r) does not affect atherosclerosis in apolipoprotein E-deficient (Apoe −/−) mice, nor does it affect IL18 cell surface binding or signaling. IL18(More)
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