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  • Peter H Duesberg, Reinhard Stindl, Ruediger Hehlmann
  • Biology, Medicine
  • Proceedings of the National Academy of Sciences…
  • 2000 (First Publication: 19 December 2000)
  • The mutation rates of cancer cells to drug and multidrug resistance are paradoxically high, i.e., 10(-3) to 10(-6), compared with those altering phenotypes of recessive genes in normal diploid cellsContinue Reading
  • Ruhong Li, A Sonik, Reinhard Stindl, David Rasnick, Peter H. Duesberg
  • Biology, Medicine
  • Proceedings of the National Academy of Sciences…
  • 2000 (First Publication: 28 March 2000)
  • For nearly a century, cancer has been blamed on somatic mutation. But it is still unclear whether this mutation is aneuploidy, an abnormal balance of chromosomes, or gene mutation. Despite enormousContinue Reading
  • Reinhard Stindl, Michael Fiegl, Heinz M Regele, Heinz Gisslinger, Martin J. Breitenseher, Christa Fonatsch
  • Biology, Medicine
  • Cancer genetics and cytogenetics
  • 1998 (First Publication: 1 November 1998)
  • Pancytopenia and fulminant disseminated intravascular coagulation in a 68-year-old woman suggested an acute hematologic malignancy. However, cytogenetic analysis on a bone marrow sample revealed aContinue Reading
  • Peter H Duesberg, Reinhard Stindl, Ruediger Hehlmann
  • Medicine, Biology
  • Proceedings of the National Academy of Sciences…
  • 2001 (First Publication: 25 September 2001)
  • Cancer cells and aneuploid cell lines can acquire resistance against multiple unrelated chemotherapeutic drugs that are over 3,000-fold those of normal levels and display spontaneous resistances upContinue Reading
  • Reinhard Stindl
  • Biology, Medicine
  • Naturwissenschaften
  • 2014 (First Publication: 1 March 2014)
  • Charles Darwin knew that the fossil record is not overwhelmingly supportive of genetic and phenotypic gradualism; therefore, he developed the core of his theory on the basis of breeding experiments.Continue Reading