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BACKGROUND Prenatal exposure to ambient PM2.5, (i.e., fine particulate matter, aerodynamic diameter ≤ 2.5 μm) has been associated with preterm birth and low birth weight. The association between prenatal PM2.5 exposure and intrauterine inflammation (IUI), an important risk factor for preterm birth and neurodevelopmental outcomes, has not been evaluated. (More)
When a comprehensive report on BPA was published in 2008, few data were available to assess the extent to which known poor glucuronidation capacity impacts BPA internal dose in infants and young children. In this paper, evidence that has emerged since the 2008 report is summarized, including: 1) human biomarker studies in children aged 0–5 years; 2) animal(More)
BACKGROUND Although previous studies suggest that exposure to traffic-related pollution during childhood increases risk of childhood overweight or obesity (COWO), the role of early-life exposure to fine particulate matter (aerodynamic diameter <2.5 micrometers, PM2.5) and its joint effect with mother's pre-pregnancy body mass index (MPBMI) on COWO remain(More)
OBJECTIVE To determine the balance of metabolism of free bisphenol A (BPA) to the inactive conjugate, BPA glucuronide (BPAG), in neonates. STUDY DESIGN Free BPA and BPAG concentrations were measured in 78 urine samples collected between December 2012 and August 2013 from a cohort of 44 healthy full term (≥ 37 weeks' gestation) neonates at 2 intervals (3-6(More)
BACKGROUND Although previous studies suggest that exposure to traffic-related pollution during childhood increases the risk of childhood overweight or obesity (COWO), the role of early life exposure to fine particulate matter (aerodynamic diameter ; ) and its joint effect with the mother's prepregnancy body mass index (MPBMI) on COWO remain unclear. (More)
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