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Traumatic brain injury in the rat: Characterization of a lateral fluid-percussion model
The role of excitatory amino acids and NMDA receptors in traumatic brain injury.
Brain injury induced by fluid percussion in rats caused a marked elevation in extracellular glutamate and aspartate adjacent to the trauma site, which contributes to delayed tissue damage after brain trauma.
Degradation of chlorinated aliphatic hydrocarbons by Methylosinus trichosporium OB3b expressing soluble methane monooxygenase
- R. Oldenhuis, R. Vink, D. Janssen, B. Witholt
- ChemistryApplied and environmental microbiology
- 1 November 1989
Degradation of trichloroethylene (TCE) by the methanotrophic bacterium Methylosinus trichosporium OB3b was studied by using cells grown in continuous culture. TCE degradation was a strictly…
Mechanisms of cerebral edema in traumatic brain injury: therapeutic developments
Attenuating blood–brain barrier permeability has become a promising approach to managing brain edema and associated swelling given that increases in cranial water content can only be derived from the vasculature.
Soluble amyloid precursor protein α reduces neuronal injury and improves functional outcome following diffuse traumatic brain injury in rats
Both estrogen and progesterone attenuate edema formation following diffuse traumatic brain injury in rats
Substance P is Associated with the Development of Brain Edema and Functional Deficits after Traumatic Brain Injury
- J. Donkin, A. Nimmo, I. Cernak, P. Blumbergs, R. Vink
- Biology, MedicineJournal of cerebral blood flow and metabolism…
- 13 May 2009
It is suggested that SP release is integrally linked to the increased vascular permeability and edema formation after brain trauma, and that treatment with an NIC, receptor antagonist reduces edema and improves neurologic outcome.
Multifunctional drugs for head injury
Red/near-infrared irradiation therapy for treatment of central nervous system injuries and disorders
A balanced précis of outcomes described in the literature regarding treatment modalities and efficacy of R/NIR-IT for injury and disease in the CNS is provided.