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Traumatic brain injury in the rat: Characterization of a lateral fluid-percussion model
View on Elsevier
The role of excitatory amino acids and NMDA receptors in traumatic brain injury.
TLDR
Brain injury induced by fluid percussion in rats caused a marked elevation in extracellular glutamate and aspartate adjacent to the trauma site, which contributes to delayed tissue damage after brain trauma.
View on PubMed
Degradation of chlorinated aliphatic hydrocarbons by Methylosinus trichosporium OB3b expressing soluble methane monooxygenase
Degradation of trichloroethylene (TCE) by the methanotrophic bacterium Methylosinus trichosporium OB3b was studied by using cells grown in continuous culture. TCE degradation was a strictly…
View on ASMUSA
Mechanisms of cerebral edema in traumatic brain injury: therapeutic developments
TLDR
Attenuating blood–brain barrier permeability has become a promising approach to managing brain edema and associated swelling given that increases in cranial water content can only be derived from the vasculature.
View on Wolters Kluwer
Soluble amyloid precursor protein α reduces neuronal injury and improves functional outcome following diffuse traumatic brain injury in rats
View on Elsevier
Both estrogen and progesterone attenuate edema formation following diffuse traumatic brain injury in rats
View on Elsevier
Substance P is Associated with the Development of Brain Edema and Functional Deficits after Traumatic Brain Injury
TLDR
It is suggested that SP release is integrally linked to the increased vascular permeability and edema formation after brain trauma, and that treatment with an NIC, receptor antagonist reduces edema and improves neurologic outcome.
View on SAGE
Multifunctional drugs for head injury
View on Springer
Red/near-infrared irradiation therapy for treatment of central nervous system injuries and disorders
TLDR
A balanced précis of outcomes described in the literature regarding treatment modalities and efficacy of R/NIR-IT for injury and disease in the CNS is provided.
View on PubMed
The neuroprotective domains of the amyloid precursor protein, in traumatic brain injury, are located in the two growth factor domains
View on Elsevier
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