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Shifting visual attention between objects and locations: evidence from normal and parietal lesion subjects.
TLDR
Space- and object-based attention components were examined in neurologically normal and parietal-lesion subjects, who detected a luminance change at 1 of 4 ends of 2 outline rectangles, indicating a spatial deficit and object deficit.
Effects of parietal injury on covert orienting of attention
TLDR
The results show that damage to the parietal lobe produces a deficit in the disengage operation when the target is contralateral to the lesion, and suggest one mechanism of the effects of parietal lesions reported in clinical neurology.
Inhibition of return : Neural basis and function
Abstract A goal of neuropsychology is to connect cognitive functions with underlying neural systems. Posner (1984; in press) has proposed a framework for doing so in which elementary mental
Saccade preparation inhibits reorienting to recently attended locations.
TLDR
Manual reaction time in normal human subjects is measured to confirm that an eccentric visual signal has a biphasic effect on covert attention and eye movements and activation of return is activated by midbrain oculomotor pathways and may function as a location "tagging" mechanism to optimize efficiency of visual search.
Spatial attention deficits in humans: a comparison of superior parietal and temporal-parietal junction lesions.
TLDR
Two groups of patients selected for lesions at the temporal-parietal junction including the superior temporal gyrus, or for lesions involving the parietal but not the inferior temporal region, performed cued-target detection tasks, suggesting that separate mechanisms mediate exogenous and endogenous processes during attention shifts.
Neural systems control of spatial orienting.
TLDR
In cases of mid-brain degeneration due to progressive supranuclear palsy, saccadic movements were abolished, while covertorienting still occurs, however, covert orienting was found to be delayed in directions in which eye movements were most affected, suggesting a role for mid- brain pathways in covert Orienting.
Toward a Functional Analysis of the Basal Ganglia
TLDR
Using the framework of equilibrium point theory of movement, it is discussed how a set switching deficit may also underlie clinical motor disturbances seen in Parkinson's disease.
How do the parietal lobes direct covert attention?
TLDR
These first experiments indicated that patients with unilateral parietal damage do worse when cued to a location in either field and then given a target in the contralesional as compared to the ipsilesional direction.
Competition between endogenous and exogenous orienting of visual attention.
TLDR
The results showed that each orienting mechanism developed its typical and independent effect in every case except for the difficult identification task.
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