Priti Kumar Roy

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1 Centre for Mathematical Biology and Ecology, Department of Mathematics, Jadavpur University, Kolkata, West Bengal 700032, India 2Department of Mathematics, Barasat College, Kolkata 700126, India 3 Department of Chemistry, Narula Institute of Technology, Kolkata 700109, India 4Agricultural and Ecological Research Unit, Indian Statistical Institute, 203 B.(More)
—Psoriasis is a frequent autoimmune chronic skin disease differentiated by T-Cells agreeable hyperpro-liferation of epidermal Keratinocytes. The feature of T-Cells held up Psoriatic scratches is the epidermal penetration of basically oligoclonal CD8 + T-Cells and also of CD4 + T-Cells in the dermis. Psoriatic lesions are sharply distinguished, red and(More)
—Psoriasis is a chronic inflammatory human skin disorder characterized by T-cell mediated hyperproliferation of Keratinocytes. The exact nature of the triggering agent of psoriasis is not yet known. Cell biological findings on the disease revealed that Helper T-cells in the human blood as well as Dendritic Cells play an important role in inflicting the(More)
Human Immunodeficiency Virus (HIV) is observed to infect the CD4+T lymphocytes in the human blood. In this article we consider a modification of a basic HIV model introduced by Herz et al. [1] representing the HIV dynamics. We introduce an intracellular delay in the disease transmission term. Our theoretical and numerical analysis show that a HIV infected(More)
—Drug therapy response of HIV-1 infected immune system is an important area of research by now. Today, a greater knowledge of drug induced CTL response allows us to better understand of cell mediated immune mechanism and humoral immune mechanism for viral infections. Moreover excessive stimulation of CTL in the immune system by different drugs leads to(More)
Entry of acquired immune deficiency syndrome virus into the host immune cell involves the participation of various components of host and viral cell unit. These components may be categorized as attachment of the viral surface envelope protein subunit, gp120, to the CD4(+) receptor and chemokine coreceptors, CCR5 and CXCR4, present on T cell surface. The(More)