Peter M. Sullivan

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Xiaolai Zhou1
Marcus B. Smolka1
Daniel H. Paushter1
Adam M. Robins1
1Xiaolai Zhou
1Marcus B. Smolka
1Daniel H. Paushter
1Adam M. Robins
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Hexanucleotide repeat expansion in the C9orf72 gene is a leading cause of frontotemporal lobar degeneration (FTLD) with amyotrophic lateral sclerosis (ALS). Reduced expression of C9orf72 has been proposed as a possible disease mechanism. However, the cellular function of C9orf72 remains to be characterized. Here we report the identification of two binding(More)
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