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Sources, Fate, and Toxic Hazards of Oxygenated Polycyclic Aromatic Hydrocarbons (PAHs) at PAH- contaminated Sites
TLDR
Oxy-PAHs are important cocontaminants that should be taken into account during risk assessment and remediation of sites with high levels of PAHs, and should be included in monitoring programs at PAH-contaminated sites, even if a number of other toxicologically relevant compounds that may also be present are not monitored. Expand
The effect of temperature and algal biomass on bacterial production and specific growth rate in freshwater and marine habitats
TLDR
Pronounced seasonality, fluctuations in nutrient quality, and variation of the grazing environment may contribute to the unexplained variation in specific growth in fresh, marine and estuarine/coastal waters. Expand
Mutagenic and carcinogenic hazards of settled house dust. I: Polycyclic aromatic hydrocarbon content and excess lifetime cancer risk from preschool exposure.
TLDR
A risk assessment revealed that exposure to PAHs at levels found in 90% of the homes would result in excess cancer risks that are considered acceptable (i.e., 1-100 x 10(-6), however, exposure to higher levels ofPAHs found in five homes yielded risks that could be higher than 1 x 10 (-4). Expand
The mutagenic hazards of settled house dust: a review.
TLDR
Risk assessment calculations performed in this study reveal that the excess cancer risks from non-dietary ingestion of carcinogenic PAHs in SHD by preschool aged children is generally in the range of what is considered acceptable (1 x 10 (-6) to 2 x 10(-6). Expand
Mutagens in contaminated soil: a review.
TLDR
The compiled data on Salmonella mutagenicity indicates significant differences in mean potency (revertents per gram dry weight) between industrial, urban, and rural/agricultural sites. Expand
Genotoxicity, cytotoxicity, and reactive oxygen species induced by single‐walled carbon nanotubes and C60 fullerenes in the FE1‐Muta™Mouse lung epithelial cells
TLDR
It is indicated that SWCNT and C60 are less genotoxic in vitro than carbon black and diesel exhaust particles and that the mutant frequency in the cII gene was unaffected by 576 hr of exposure. Expand
Air pollution induces heritable DNA mutations
TLDR
The results indicate that human and wildlife populations in proximity to integrated steel mills may be at risk of developing germline mutations more frequently because of the inhalation of airborne chemical mutagens. Expand
Increased mutant frequency by carbon black, but not quartz, in the lacZ and cII transgenes of muta™mouse lung epithelial cells
Carbon black and quartz are relatively inert solid particulate materials that are carcinogenic in laboratory animals. Quartz is a human carcinogen, whereas data on carbon black are contradictory, andExpand
The mutagenic hazards of aquatic sediments: a review.
TLDR
A comprehensive review of published studies that investigated the genotoxicity of sediments in rivers, lakes and marine habitats revealed mutagenic potency values for sediment extracts that spans over seven orders of magnitude from not detectable to highly potent. Expand
Heritable reproductive effects of benzo[a]pyrene on the fathead minnow (Pimephales promelas)
TLDR
This study investigated the survivorship of fathead minnow larvae two generations removed from an exposure to the potent mutagen benzo[a]pyrene and found that reproductive capacity and larval survivorship were significantly lower than the solvent control. Expand
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