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Applied Blood Group Serology
Erythrocyte autoantibodies in paediatric patients with sickle cell disease receiving transfusion therapy: frequency, characteristics and significance
TLDR
Formation of warm erythrocyte autoantibodies in association with transfusions is not rare in paediatric patients with sickle cell disease, but Clinicians should be aware of this complication and recognize that the presence of surface C3 is often associated with significant haemolysis.
A proposal to standardize terminology for weak D antigen
TLDR
Although the presence of the organism in blood causes frank transfusion reactions, no fatalities have been reported and it may be possible to determine whether E. agglomerans is an opportunistic contaminant that causes few sequelae, or whether its presence has more farreaching effects.
ISBT Working Party on Terminology for Red Cell Surface Antigens: Munich Report
TLDR
This report is a compilation of the recommendations made by the subcommittees and accepted by the Working Party at the Budapest meeting on August lst, 1982 and at the Munich meeting on July 21st, 1984.
Anti‐Wrb, and Other Autoantibodies Responsible for Positive Direct Antiglobulin Tests in 150 Individuals
TLDR
The full specificities of autoantibodies, other than anti‐Wrb and anti‐dl, in the 150 patients are described, as are the natures of the protein red cell coatings that caused the positive direct antiglobulin tests.
Terminology for blood group antigens and genes—historical origins and guidelines in the new millennium
TLDR
There were problems with ABO terminology during the several decades following Landsteiner’s discovery because of two different terminologies, the CcDdEe terms of Fisher and Race12 and Race and Sanger13 and the Rh/Hr terms used by Wiener and his followers.
Monocyte phagocytic activity in sickle cell disease.
TLDR
Monocytes from sickle cell disease patients during vasoocclusive crises demonstrated increased phagocytic activity compared to the normal controls, and this activity is not deficient and is not a factor in the predisposition to infections.
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