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The emerging role for chemokines in epilepsy
Immunomodulatory Effects of Etanercept in an Experimental Model of Spinal Cord Injury
Treatment with etanercept reduces the development of inflammation and tissue injury events associated with spinal cord trauma and it is clearly demonstrated that TNF-α inhibitor significantly ameliorated the recovery of limb function.
Acute metabolic brain changes following traumatic brain injury and their relevance to clinical severity and outcome
Spectroscopic measures of neuro-axonal damage occurring soon after a brain trauma are clinically relevant and can represent a reliable index of injury severity and disease outcome in patients with TBI.
Entangling COVID-19 associated thrombosis into a secondary antiphospholipid antibody syndrome: Diagnostic and therapeutic perspectives (Review)
It is proposed here that the thromboembolic events and eventually the development of DIC provoked by SARS-CoV-2 infection may represent a secondary anti-phospholipid antibody syndrome (APS), and it will be proved that secondary APS is likely responsible for coagulopathy during the course of COVID-19 infection.
Effects of Palmitoylethanolamide on Signaling Pathways Implicated in the Development of Spinal Cord Injury
It is indicated that PEA reduces inflammation and tissue injury associated with SCI and suggest a regulatory role for endogenous PPAR-α signaling in the inflammatory response associated with spinal cord trauma.
Predictive Biomarkers of Recovery in Traumatic Brain Injury
The various factors that must be taken into account in the search for a reliable non-invasive biomarkers in traumatic brain injury and their role in the diagnosis and outcome evaluation are examined.
NMDA receptor antagonist felbamate reduces behavioral deficits and blood-brain barrier permeability changes after experimental subarachnoid hemorrhage in the rat.
It is demonstrated that, in terms of behavioral and microvascular effects, FBM is beneficial in a dose-dependent manner after experimental SAH in rats, and reinforces the concept that NMDA excitotoxicity is involved in the cerebral dysfunction that follows SAH.
Absence of endogenous interleukin‐10 enhances secondary inflammatory process after spinal cord compression injury in mice
The presence of IL‐10 reduces the development of inflammation and tissue injury events associated with spinal cord trauma and the genetic absence ofIL‐10 worsened the recovery of limb function when compared with IL‐ 10 wild‐type mice group.