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Tissue displacement and impact force are important contributors to outcome after spinal cord contusion injury

• Nader Ghasemlou, Bradley J Kerr, Samuel Paul David
• Experimental Neurology
• 2005

Spinal cord contusion injury in rodents is widely used as a model for spinal cord trauma in humans. Several biomechanical variables can influence injury outcome. In this work, we have assessed the… (More)

Mitogen-activated protein kinase-activated protein kinase 2 (MK2) contributes to secondary damage after spinal cord injury.

• Nader Ghasemlou, Ruben López-Vales, +4 authors Samuel Paul David
• The Journal of neuroscience : the official…
• 2010

The inflammatory response contributes importantly to secondary tissue damage and functional deficits after spinal cord injury (SCI). In this work, we identified mitogen-activated protein kinase… (More)

Inhibition of the Ca²⁺-dependent K⁺ channel, KCNN4/KCa3.1, improves tissue protection and locomotor recovery after spinal cord injury.

• Delphine Bouhy, Nader Ghasemlou, +4 authors Samuel Paul David
• The Journal of neuroscience : the official…
• 2011

Spinal cord injury (SCI) triggers inflammatory responses that involve neutrophils, macrophages/microglia and astrocytes and molecules that potentially cause secondary tissue damage and functional… (More)

Phospholipase A2 superfamily members play divergent roles after spinal cord injury.

• Ruben López-Vales, Nader Ghasemlou, +11 authors Samuel Paul David
• FASEB journal : official publication of the…
• 2011

Spinal cord injury (SCI) results in permanent loss of motor functions. A significant aspect of the tissue damage and functional loss may be preventable as it occurs, secondary to the trauma. We show… (More)

Fenretinide promotes functional recovery and tissue protection after spinal cord contusion injury in mice.

• Ruben López-Vales, Adriana Redensek, +6 authors Samuel Paul David
• The Journal of neuroscience : the official…
• 2010

The inflammatory response is thought to contribute to secondary damage after spinal cord injury (SCI). Polyunsaturated fatty acids (PUFAs) play an important role in the onset and resolution of… (More)

Inflammatory pathways in spinal cord injury.

• Samuel Paul David, Juan Guillermo Zarruk, Nader Ghasemlou
• International review of neurobiology
• 2012

Injury to the spinal cord results in direct damage to axons, neuronal cell bodies, and glia that cause functional loss below the site of injury. In addition, the injury also triggers an inflammatory… (More)

The protective effects of 15-deoxy-delta-(12,14)-prostaglandin J2 in spinal cord injury.

• Bradley J Kerr, Elizabeth I. Girolami, Nader Ghasemlou, Suh Young Jeong, Samuel Paul David
• Glia
• 2008

Secondary tissue damage that occurs within days after spinal cord injury contributes significantly to permanent paralysis, sensory loss, and other functional disabilities. The acute inflammatory… (More)

5,6-EET is released upon neuronal activity and induces mechanical pain hypersensitivity via TRPA1 on central afferent terminals.

• Marco Sisignano, Chul-Kyu Park, +16 authors Christian Brenneis
• The Journal of neuroscience : the official…
• 2012

Epoxyeicosatrienoic acids (EETs) are cytochrome P450-epoxygenase-derived metabolites of arachidonic acid that act as endogenous signaling molecules in multiple biological systems. Here we have… (More)

Phenotyping the function of TRPV1-expressing sensory neurons by targeted axonal silencing.

• Christian Brenneis, Katrin Kistner, +12 authors Clifford J Woolf
• The Journal of neuroscience : the official…
• 2013

Specific somatosensations may be processed by different subsets of primary afferents. C-fibers expressing heat-sensitive TRPV1 channels are proposed, for example, to be heat but not mechanical pain… (More)

T cells contribute to lysophosphatidylcholine-induced macrophage activation and demyelination in the CNS.

• Nader Ghasemlou, Suh Young Jeong, Steve Lacroix, Samuel Paul David
• Glia
• 2007

We have previously shown that intraspinal microinjection of lysophosphatidylcholine (LPC), a potent demyelinating agent, results in a rapid but brief influx of T cells (between 6 and 12 h). This is… (More)

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