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Cognition and aphasia: a discussion and a study.

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Empty speech in Alzheimer's disease and fluent aphasia.

TLDR
Patients with Alzheimer's dementia were distinguished from patients with Wernicke's aphasia by producing more empty phrases and conjunctions, whereas patients with EMTs with anomic aphasias produced more neologisms, and verbal and literal paraphasias.
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Manual of Aphasia and Aphasia Therapy

TLDR
Manual of aphasia and aphasIA therapy / , Manual of a speech-language-language therapy /, and more recently “Tests for language impairment in children with a history of abuse".

Improved naming after TMS treatments in a chronic, global aphasia patient – case report

We report improved ability to name pictures at 2 and 8 months after repetitive transcranial magnetic stimulation (rTMS) treatments to the pars triangularis portion of right Broca’s homologue in a 57…
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Aphasia with predominantly subcortical lesion sites: description of three capsular/putaminal aphasia syndromes.

TLDR
Nine cases of subcortical aphasia with capsular/putaminal (C/P) lesion sites demonstrated on computed tomographic (CT) scans did not completely resemble cases of either Broca's, Wernicke's, global, or thalamic aphasias in neurologic findings, CT scan lesion Sites, or language behavior.
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C-Speak Aphasia alternative communication program for people with severe aphasia: Importance of executive functioning and semantic knowledge

TLDR
Cognitive and linguistic factors relative to how they affected individual patients' ability to communicate expressively using C-Speak Aphasia, an alternative communication computer program that is primarily picture-based, were explored.
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Stuttering following acquired brain damage: A review of the literature

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Using a computer to communicate: Effect of executive function impairments in people with severe aphasia

Background : Some individuals with severe non-fluent aphasia do not respond in a functional way to any form of communication therapy. Others show improved ability to communicate with treatment…
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Foreign accent syndrome due to conversion disorder: Phonetic analyses and clinical course

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Severe nonfluency in aphasia. Role of the medial subcallosal fasciculus and other white matter pathways in recovery of spontaneous speech.

TLDR
It is suggested that one explanation for the more severe limitation in spontaneous speech in the first group is the extensive white matter lesion in these two subcortical pathways had interrupted a large number of connections for initiation and preparation of speech movements and limbic aspects of speech.
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