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Regulation of calcium fluxes in pancreatic islets: the role of membrane depolarization.
TLDR
It is concluded that K+-induced plasma membrane depolarization reproduces the effect of glucose to stimulate but not to inhibit 45Ca efflux from perifused islets. Expand
Regulation of calcium fluxes in rat pancreatic islets: the role of K+ conductance.
TLDR
It is concluded that TEA by inhibiting K+ conductance mimics to a limited extent the effect of glucose in causing the gating of voltage-sensitive calcium channels in isolated islets. Expand
Regulation of calcium fluxes in rat pancreatic islets. Quinine mimics the dual effect of glucose on calcium movements.
TLDR
Quinine reproduces the effect of glucose to activate voltage-sensitive calcium channels and to stimulate the entry of calcium into the B-cell, and the glucose-induced inhibition of calcium outflow rate does not appear to be mediated by changes in K+ conductance. Expand
Quantitative Determination of Acenocoumarin in Anticoagulated Patients
Abstract A new procedure of pre-concentration on Tenax GC followed by high performance liquid chromatography analysis has been developed for the quantitative determination of acenocoumarin in humanExpand
Parameters for evaluation of lipid metabolism.
TLDR
Regular monitoring of plasma triglyceride, cholesterol, and phospholipid concentrations during and after cessation of fat infusion is recommended for each patient who is receiving daily fat infusions so that the safe rate of infusion for that individual can be determined. Expand
In vitro metabolism of a new 4-hydroxycoumarin anticoagulant. Structure of an unusual metabolite.
The metabolism of clocoumarol, 3-[1-[P-(2-chlorethyl)phenyl]butyl]-4-hydroxycoumarin, by rat liver microsomes was investigated. The chemical structure of the main metabolite isExpand
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