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Cytidine Deamination of Retroviral DNA by Diverse APOBEC Proteins
Specific Expression of Activation-induced Cytidine Deaminase (AID), a Novel Member of the RNA-editing Deaminase Family in Germinal Center B Cells*
Findings suggest that AID is a new member of the RNA-editing deaminase family and may play a role in genetic events in the germinal center B cell.
Fructose transporter in human spermatozoa and small intestine is GLUT5.
Molecular cloning of an apolipoprotein B messenger RNA editing protein.
A full-length complementary DNA clone encoding an apo B messenger RNA editing protein (REPR) was isolated from rat small intestine and may lead to the identification of other eukaryotic RNA editing proteins.
Role of the gut in lipid homeostasis.
An integrative view of intestinal lipid homeostasis is provided through recent findings on the role of lipid flux and fatty acid signaling via diverse receptor pathways in regulating absorption and production of satiety factors.
Decreased Hepatic Triglyceride Accumulation and Altered Fatty Acid Uptake in Mice with Deletion of the Liver Fatty Acid-binding Protein Gene*
Data point to an inducible defect in fatty acid utilization in fasted L-Fabp–/– mice that involves targeting of substrate for use in triglyceride metabolism.
CD36 deficiency impairs intestinal lipid secretion and clearance of chylomicrons from the blood.
It is concluded that CD36 is important for both secretion and clearance of intestinal lipoproteins, and may constitute a risk factor for diet-induced type 2 diabetes and cardiovascular disease.
IRE1α-XBP1s induces PDI expression to increase MTP activity for hepatic VLDL assembly and lipid homeostasis.
The forkhead box M1 transcription factor contributes to the development and growth of mouse colorectal cancer.
- Y. Yoshida, I. Wang, Helena M Yoder, N. Davidson, R. Costa
- Biology, MedicineGastroenterology
- 1 April 2007
It is suggested that Foxm1 is critical for the proliferation and growth of colorectal cancer.
APOLIPOPROTEIN B: mRNA editing, lipoprotein assembly, and presecretory degradation.
Although control of lipid secretion in vivo is primarily achieved at the level of lipoprotein particle size, regulation of apoB production by presecretory degradation may be relevant in some dyslipidemic states.