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Cellular localization of AT1 receptor mRNA and protein in normal placenta and its reduced expression in intrauterine growth restriction. Angiotensin II stimulates the release of vasorelaxants.
  • X. Li, M. Shams, A. Ahmed
  • Biology, Medicine
    The Journal of clinical investigation
  • 15 January 1998
TLDR
Observations suggest the existence of a cross-talk between AT1 or AT2 receptors in trophoblast and that the reduction in placental AT1 receptors in IUGR may, in part, account for poor placental function in this disorder.
Is central hyperacusis a symptom of 5-hydroxytryptamine (5-HT) dysfunction?
TLDR
It is proposed that 5-HT dysfunction is a probable cause of increased auditory sensitivity manifested as centralhyperacusis or phonophobia, and a separate aetiology of central hyperacusis is therefore proposed, with a symptom profile distinct from the peripheral hyperacusIS.
Neuronal 5-HT Receptors and SERT
TLDR
5-hydroxytryptamine (5-HT, serotonin) is an ancient biochemical manipulated through evolution to be utilized extensively throughout the animal and plant kingdoms, and also as a local hormone in numerous other tissues, including the gastrointestinal tract, the cardiovascular system and immune cells.
Ability of angiotensin II to modulate striatal dopamine release via the AT1 receptor in vitro and in vivo
TLDR
Direct evidence is provided that angiotensin II acting via the AT1 receptor subtype facilitates the release of dopamine in the rat striatum in vitro and in vivo.
5-HT3 receptors mediate inhibition of acetylcholine release in cortical tissue
TLDR
Evidence that a reduction in cortical cholinergic function can be effected in vitro by 5-HT3 receptors is provided and Radioligand binding studies show a high density of 5- HT3 receptors in theCholinergic-rich entorhinal cortex.
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