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Nitric oxide improves copper tolerance via modulation of superoxide and hydrogen peroxide levels. This reflects the necessity of a well-coordinated interplay between NO and ROS during stress tolerance. Copper (Cu) excess causes toxicity and one probable consequence of this is the disturbance of cell redox state maintenance, inter alia, by reactive oxygen-(More)
Plants are able to dynamically adapt to their environment by reprogramming of their growth and development. Copper (Cu2+) excess modifies shoot and root architecture of plants by a lesser known mechanism, therefore the involvement of a major hormone component (auxin) and a signal molecule (nitric oxide) in Cu2+-induced morphological responses were studied(More)
Selenium (Se) phytotoxicity at the cellular level disturbs the synthesis and functions of proteins, together with the generation of an oxidative stress condition. This study reveals the nitro-oxidative stress events, supplemented by a broad spectrumed characterisation of the Se-induced symptoms. Applying several, carefully selected methods, we investigated(More)
Selenite oppositely modifies cytokinin and nitric oxide metabolism in Arabidopsis organs. A mutually negative interplay between the molecules exists in selenite-exposed roots; and their overproduction causes selenite insensitivity. Selenium-induced phytotoxicity is accompanied by developmental alterations such as primary root (PR) shortening. Growth changes(More)
Copper (Cu) in excess can disturb the cell redox status maintained by reactive oxygen- (ROS) and nitrogen species. With the help of the nitric oxide (NO)-deficient nia1nia2noa1-2 mutant, the role of NO in copper stress tolerance and its relationship with ROS was examined. Under control conditions and also during Cu exposure, the NO level in the cotyledon(More)
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